A Mechanism for Health Impacts of Electromagnetic Fields | Martin Pall

Finally an established mechanism for health impacts of electromagnetic fields. How diverse health effects are produced by downstream consequences of this mechanism.

Martin Pall

26 different studies have each shown that microwave/ lower frequency electromagnetic fields (EMFs) produce biological effects via activation of voltage-gated calcium channels (VGCCs). Forces of such EMFs on the voltage sensor controlling the VGCCs are approximately 7.2 million times stronger than the forces on singly charged groups in the aqueous phases of the cell. This predicts, in turn, that the current safety guidelines allow us to be exposed to EMFs that are approximately 7.2 million times too high.

The downstream effects of VGCC activation act via elevated nitric oxide signaling, peroxynitrite/free radical effects and excessive [Ca2+]i signaling produce the various well documented effects of EMF exposure including: Oxidative stress; cellular DNA damage; cancer; massive impacts of the brain leading to widespread neuropsychiatric effects; changes in endocrine (hormone) effects; lowered male and female fertility, increased spontaneous abortion; cardiac effects including arrhythmias, tachycardia, bradycardia and heart palpitations; melatonin depletion and sleep deprivation; EHS; breakdown of the blood-brain barrier. The extraordinary cellular DNA damage produced by cell phone-like pulsed microwave EMF exposures (Schwarz et al) as compared with that produced by ionizing radiation can be understood as being due to the three levels of amplification leading from VGCC activation to free radical production with the free radicals producing, in turn, the DNA damage. The industry propaganda claim that ionizing radiation is dangerous but non-ionizing radiation is not, is exactly opposite from the actual findings on cellular DNA damage!

Five worse case scenarios are discussed. Ever increasing EMF exposures are predicted to lead to a crash in human reproduction; DNA damage in germ line cells are producing large mutational increases; widespread neuropsychiatric effects will become increasingly severe; VGCC activation, known to cause the changes in synaptic development and function, will produce increasingly common autism in future generations; the known role of excessive [Ca2+]i in Alzheimer’s and other neurodegenerative diseases may be the the primary cause of circa age 30 dementias and may be leading to very large increases in very early onset dementias. Combined effects of these may be expected produce massive impacts on human populations.

Bio: Martin L. Pall, Professor Emeritus of Biochemistry and Basic Medical Sciences, Washington State University. martin_pall@wsu.edu

Recorded at the Society for Scientific Exploration conference at Yale University, 2017

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Published on December 19, 2018